CORDIS Project
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This project investigates the role of the SLFN2 gene in maintaining the quiescent state of T-cells in the immune system. By understanding how quiescence is regulated, the research aims to improve immune responses and develop therapies for immune disorders.
The immune system maintains a vast repertoire of lymphocytes waiting to respond to microbial invasion.
These cells are kept in a quiescent state, characterized by arrest in G0 and a decrease in cell size and metabolic activity, until they are activated.
Not long ago, quiescence was considered equivalent to “absence of activation”, namely a default state of the cell.
Recently our discovery of a chemically induced mutation, elektra, enabled us to illustrate what happens when quiescence fails.
Elek…
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