CORDIS Project
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This project investigates the roles of interleukin-6 and interleukin-1 beta in the response to exercise in patients with type 2 diabetes. By using innovative mouse models and clinical approaches, it aims to optimize exercise therapy for all patients, particularly those who do not respond well to traditional methods.
Hyperglycaemia in type 2 diabetes (T2D) doubles mortality and arises due to failure of pancreatic β-cell insulin secretory function to overcome insulin resistance.
Exercise reduces hyperglycaemia by improving insulin sensitivity and β-cell function; however, the variability of this beneficial effect is huge and is associated with high blood glucose and poor insulin secretion.
Hyperglycaemia increases interleukin-(IL)1β levels causing β-cell death and insulin secretory dysfunction (glucotoxicity)…
THE UNIVERSITY OF BIRMINGHAM
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