CORDIS Project
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This project investigates the mechanisms by which herpes simplex virus-1 generates mutations during infection. It aims to identify host and viral proteins that regulate viral genome expression and replication, potentially revealing new insights into herpesvirus biology and virology.
Infection with herpes simplex virus-1 (HSV-1) can lead to diverse outcomes ranging from asymptomatic infection to life threatening encephalitis.
Emergence of drug resistant viral isolates, leading to treatment failure, is caused by rapid generation of mutations.
For DNA viruses (such as herpesviruses) it is assumed that recombination and complementation can provide important mechanisms for generating and maintaining genome diversity.
We therefore suggest that the phenomenon (We recently identifi…
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