CORDIS Project
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This project investigates how microglia, the brain's immune cells, interact with synapses in Alzheimer's disease compared to healthy brains. By using advanced techniques, it aims to identify specific proteins involved in these interactions, which could lead to new treatment targets for Alzheimer's disease.
Microglia are immune cells that monitor our brain and degrade unhealthy neurons and excess synapses.
In Alzheimer’s disease (AD) however, microglia excessively destroy synapses, marking them as enticing treatment targets. AD patients accumulate cortical Amyloid-beta (Aβ) plaques and neurofibrillary tau tangles, whilst losing neurons and synapses, and consequently: memory and cognition.
Microglia are the brain’s immune cells, mediate neuroinflammation, continuously monitor their surrounding cells…
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