CORDIS Project
Funding breakdown and partner intelligence are Premium
Sign in and upgrade to Premium for EU contribution totals, consortium analytics, OpenAlex research context, and AI summaries. · 0 consortium intelligence fields visible of 1
Start free • Cancel anytime • 14-day refund guarantee
This project investigates the metabolic dysfunctions caused by defects in mitochondrial electron transport chains. It aims to identify key genes and pathways involved in these dysfunctions, focusing on how they contribute to cellular and tissue deterioration, particularly in the brain.
Mitochondria are essential organelles that carry out critical metabolic functions within the cells.
Electron transport chain (ETC) defects occurring from mitochondrial disease mutations compromise cellular fitness and survival.
This biochemical failure is thought to underlie pathologies associated with mitochondrial dysfunction.
However, the precise metabolic processes, signaling pathways and compensatory responses resulting from a defective mitochondrial ETC that drive these fatal disorders are…
UNIVERSIDAD AUTONOMA DE MADRID
Partner organizations (coordinator is shown above), with normalized type and CORDIS activity type. Guests see up to 4 partners.
Similar projects, consortium collaboration history, frequent partners, and OpenAlex research context.
Guests see up to 5 EuroSciVoc fields.
Guests see up to 5 topics.
Guests see up to 5 keywords.
