CORDIS Project
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MONSil-AD investigates the mechanisms behind neuronal silencing in Alzheimer's disease using mouse models. The project focuses on understanding how tau accumulation and microglial factors contribute to this dysfunction, with the goal of developing early detection tools and targeted therapies.
Alzheimer’s disease (AD) is defined by pathological brain changes, including the accumulation of β-amyloid and tau deposits over decades.
In addition, data in human patients and rodent models indicate that neuronal dysfunctions, i.e., altered action potential firing rates, are further hallmarks of AD.
Two forms of neuronal dysfunctions exist - neuronal hyperactivity at early stages, followed later by neuronal silencing.
The causes of neuronal hyperactivity have been widely studied.
However, the…
KLINIKUM RECHTS DER ISAR DER TECHNISCHEN UNIVERSITAT MUNCHEN
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