CORDIS Project
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This project investigates how alternative splicing of the myosin VI gene influences cancer cell behavior, particularly in cell migration and adaptation. By identifying splicing regulators and their roles, the research aims to uncover the molecular mechanisms behind splicing changes during cancer progression.
Alternative splicing (AS), by affecting nearly 90 percent of human genes, is the major contributor to protein diversity.
Mutations that disrupt AS regulatory sequences are a widespread source of human diseases.
Various evidences suggest that an AS reprogramming is critical in cell adaptation during cancer development.
In spite of increasing interest in the potential oncogenic role of AS, little is known about the molecular mechanisms behind its deregulation during the somatic to the cancer cell…
IFOM-ISTITUTO FONDAZIONE DI ONCOLOGIA MOLECOLARE ETS
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