CORDIS Project
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This research investigates the role of intracellular trafficking in the accumulation of beta-amyloid in neurons, a key factor in Alzheimer's disease. It aims to understand how alterations in trafficking mechanisms contribute to neurodegeneration and memory impairment.
In neurons of Alzheimer’s disease (AD) there is an aberrant accumulation of beta-amyloid (Aβ) at synapses that renders difficult the formation of new memories for AD patients.
Intracellular trafficking abnormalities have been implicated in Aβ accumulation.
This research project aims to define how neuronal intracellular trafficking is mechanistically involved in Aβ accumulation that leads to AD.
We will determine how the intracellular itinerary of the amyloid precursor protein in neurons influen…
FACULDADE DE CIENCIAS MEDICAS DA UNIVERSIDADE NOVA DE LISBOA
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